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Artigo de periodico
Deletion of OGG1 results in a differential signature of oxidized purine base damage in mtDNA regions (2019)
Chimienti, Guglielmina ; Pesce, Vito; Fracasso, Flavio; Russo, Francesco; Souza-Pinto, Nadja Cristhina de ; Bohr, Vilhelm A; Lezza, Angela Maria Serena
Source: International Journal of Molecular Sciences. Unidade: IQ
Subjects: DANO AO DNA, ESTRESSE OXIDATIVO, MITOCÔNDRIAS
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ABNT
CHIMIENTI, Guglielmina et al. Deletion of OGG1 results in a differential signature of oxidized purine base damage in mtDNA regions. International Journal of Molecular Sciences, v. 20, n. 13, p. 1-11, 2019Tradução . . Disponível em: https://doi.org/10.3390/ijms20133302. Acesso em: 28 abr. 2024.
APA
Chimienti, G., Pesce, V., Fracasso, F., Russo, F., Souza-Pinto, N. C. de, Bohr, V. A., & Lezza, A. M. S. (2019). Deletion of OGG1 results in a differential signature of oxidized purine base damage in mtDNA regions. International Journal of Molecular Sciences, 20( 13), 1-11. doi:10.3390/ijms20133302
NLM
Chimienti G, Pesce V, Fracasso F, Russo F, Souza-Pinto NC de, Bohr VA, Lezza AMS. Deletion of OGG1 results in a differential signature of oxidized purine base damage in mtDNA regions [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 13): 1-11.[citado 2024 abr. 28 ] Available from: https://doi.org/10.3390/ijms20133302
Vancouver
Chimienti G, Pesce V, Fracasso F, Russo F, Souza-Pinto NC de, Bohr VA, Lezza AMS. Deletion of OGG1 results in a differential signature of oxidized purine base damage in mtDNA regions [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 13): 1-11.[citado 2024 abr. 28 ] Available from: https://doi.org/10.3390/ijms20133302
Artigo de periodico
Metabolism, genomics, and DNA repair in the mouse aging liver (2011)
Lebel, Michel; Souza-Pinto, Nadja Cristhina de ; Bohr, Vilhelm A
Source: Current Gerontology and Geriatrics Research. Unidade: IQ
Subjects: DANO AO DNA, METABOLISMO, GENOMAS
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ABNT
LEBEL, Michel e SOUZA-PINTO, Nadja Cristhina de e BOHR, Vilhelm A. Metabolism, genomics, and DNA repair in the mouse aging liver. Current Gerontology and Geriatrics Research, p. 1-15, 2011Tradução . . Disponível em: https://doi.org/10.1155/2011/859415. Acesso em: 28 abr. 2024.
APA
Lebel, M., Souza-Pinto, N. C. de, & Bohr, V. A. (2011). Metabolism, genomics, and DNA repair in the mouse aging liver. Current Gerontology and Geriatrics Research, 1-15. doi:10.1155/2011/859415
NLM
Lebel M, Souza-Pinto NC de, Bohr VA. Metabolism, genomics, and DNA repair in the mouse aging liver [Internet]. Current Gerontology and Geriatrics Research. 2011 ; 1-15.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1155/2011/859415
Vancouver
Lebel M, Souza-Pinto NC de, Bohr VA. Metabolism, genomics, and DNA repair in the mouse aging liver [Internet]. Current Gerontology and Geriatrics Research. 2011 ; 1-15.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1155/2011/859415
Artigo de periodico
Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane (2010)
Aamann, Maria D; Sorensen, Martin M; Hvitby, Christina; Berquist, Brian R; Muftuoglu, Meltem; Tian, Jingyan; Souza-Pinto, Nadja Cristhina de ; Scheibye-Knudsen, Morten; Wilson III, David M; Stevnsner, Tinna V.; Bohr, Vilhelm A
Source: The Faseb Journal. Unidade: IQ
Subjects: ESTRESSE OXIDATIVO, DNA MITOCONDRIAL, DOENÇAS NEURODEGENERATIVAS
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ABNT
AAMANN, Maria D et al. Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane. The Faseb Journal, v. 24, n. 7, p. 2334-2346, 2010Tradução . . Disponível em: https://doi.org/10.1096/fj.09-147991. Acesso em: 28 abr. 2024.
APA
Aamann, M. D., Sorensen, M. M., Hvitby, C., Berquist, B. R., Muftuoglu, M., Tian, J., et al. (2010). Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane. The Faseb Journal, 24( 7), 2334-2346. doi:10.1096/fj.09-147991
NLM
Aamann MD, Sorensen MM, Hvitby C, Berquist BR, Muftuoglu M, Tian J, Souza-Pinto NC de, Scheibye-Knudsen M, Wilson III DM, Stevnsner TV, Bohr VA. Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane [Internet]. The Faseb Journal. 2010 ; 24( 7): 2334-2346.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1096/fj.09-147991
Vancouver
Aamann MD, Sorensen MM, Hvitby C, Berquist BR, Muftuoglu M, Tian J, Souza-Pinto NC de, Scheibye-Knudsen M, Wilson III DM, Stevnsner TV, Bohr VA. Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane [Internet]. The Faseb Journal. 2010 ; 24( 7): 2334-2346.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1096/fj.09-147991
Artigo de periodico
Base excision repair activities differ in human lung cancer cells and corresponding normal controls (2010)
Karahalil, Bensu; Bohr, Vilhelm A; Souza-Pinto, Nadja Cristhina de
Source: Anticancer Research. Unidade: IQ
Subjects: DANO AO DNA, NEOPLASIAS DO TRATO RESPIRATÓRIO
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ABNT
KARAHALIL, Bensu e BOHR, Vilhelm A e SOUZA-PINTO, Nadja Cristhina de. Base excision repair activities differ in human lung cancer cells and corresponding normal controls. Anticancer Research, v. 30, n. 12, p. 4963-4972, 2010Tradução . . Disponível em: http://ar.iiarjournals.org/content/30/12/4963.full.pdf+html. Acesso em: 28 abr. 2024.
APA
Karahalil, B., Bohr, V. A., & Souza-Pinto, N. C. de. (2010). Base excision repair activities differ in human lung cancer cells and corresponding normal controls. Anticancer Research, 30( 12), 4963-4972. Recuperado de http://ar.iiarjournals.org/content/30/12/4963.full.pdf+html
NLM
Karahalil B, Bohr VA, Souza-Pinto NC de. Base excision repair activities differ in human lung cancer cells and corresponding normal controls [Internet]. Anticancer Research. 2010 ; 30( 12): 4963-4972.[citado 2024 abr. 28 ] Available from: http://ar.iiarjournals.org/content/30/12/4963.full.pdf+html
Vancouver
Karahalil B, Bohr VA, Souza-Pinto NC de. Base excision repair activities differ in human lung cancer cells and corresponding normal controls [Internet]. Anticancer Research. 2010 ; 30( 12): 4963-4972.[citado 2024 abr. 28 ] Available from: http://ar.iiarjournals.org/content/30/12/4963.full.pdf+html
Artigo de periodico
Mitochondrial helicases and mitochondrial genome maintenance (2010)
Souza-Pinto, Nadja Cristhina de ; Aamann, Maria D; Kulikowicz, Tomasz; Stevnsner, Tinna V; Bohr, Vilhelm A
Source: Mechanisms of Ageing and Development. Unidade: IQ
Subjects: DNA MITOCONDRIAL, BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA-PINTO, Nadja Cristhina de et al. Mitochondrial helicases and mitochondrial genome maintenance. Mechanisms of Ageing and Development, v. 131, n. 7-8, p. 503-510, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.mad.2010.04.009. Acesso em: 28 abr. 2024.
APA
Souza-Pinto, N. C. de, Aamann, M. D., Kulikowicz, T., Stevnsner, T. V., & Bohr, V. A. (2010). Mitochondrial helicases and mitochondrial genome maintenance. Mechanisms of Ageing and Development, 131( 7-8), 503-510. doi:10.1016/j.mad.2010.04.009
NLM
Souza-Pinto NC de, Aamann MD, Kulikowicz T, Stevnsner TV, Bohr VA. Mitochondrial helicases and mitochondrial genome maintenance [Internet]. Mechanisms of Ageing and Development. 2010 ; 131( 7-8): 503-510.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.mad.2010.04.009
Vancouver
Souza-Pinto NC de, Aamann MD, Kulikowicz T, Stevnsner TV, Bohr VA. Mitochondrial helicases and mitochondrial genome maintenance [Internet]. Mechanisms of Ageing and Development. 2010 ; 131( 7-8): 503-510.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.mad.2010.04.009
Artigo de periodico
The mitochondrial transcription factor A functions in mitochondrial base excision repair (2010)
Canugovi, Chandrika; Maynard, Scott; Bayne, Anne-Cécile V; Sykora, Peter; Tian, Jingyan; Souza-Pinto, Nadja Cristhina de ; Croteau, Deborah L; Bohr, Vilhelm A
Source: DNA Repair. Unidade: IQ
Subjects: MITOCÔNDRIAS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CANUGOVI, Chandrika et al. The mitochondrial transcription factor A functions in mitochondrial base excision repair. DNA Repair, v. 9, n. 10, p. 1080-1089 : + Suplementary Materials, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2010.07.009. Acesso em: 28 abr. 2024.
APA
Canugovi, C., Maynard, S., Bayne, A. -C. V., Sykora, P., Tian, J., Souza-Pinto, N. C. de, et al. (2010). The mitochondrial transcription factor A functions in mitochondrial base excision repair. DNA Repair, 9( 10), 1080-1089 : + Suplementary Materials. doi:10.1016/j.dnarep.2010.07.009
NLM
Canugovi C, Maynard S, Bayne A-CV, Sykora P, Tian J, Souza-Pinto NC de, Croteau DL, Bohr VA. The mitochondrial transcription factor A functions in mitochondrial base excision repair [Internet]. DNA Repair. 2010 ; 9( 10): 1080-1089 : + Suplementary Materials.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.dnarep.2010.07.009
Vancouver
Canugovi C, Maynard S, Bayne A-CV, Sykora P, Tian J, Souza-Pinto NC de, Croteau DL, Bohr VA. The mitochondrial transcription factor A functions in mitochondrial base excision repair [Internet]. DNA Repair. 2010 ; 9( 10): 1080-1089 : + Suplementary Materials.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.dnarep.2010.07.009
Artigo de periodico-carta/editorial
Mitochondria and aging (Editorial) (2010)
Prolla, Tomas A; Bohr, Vilhelm A; Souza-Pinto, Nadja Cristhina de
Source: Mechanisms of Ageing and Development. Unidade: IQ
Subjects: MITOCÔNDRIAS, DNA MITOCONDRIAL
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ABNT
PROLLA, Tomas A e BOHR, Vilhelm A e SOUZA-PINTO, Nadja Cristhina de. Mitochondria and aging (Editorial). Mechanisms of Ageing and Development. Clare: Instituto de Química, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.mad.2010.07.002. Acesso em: 28 abr. 2024. , 2010
APA
Prolla, T. A., Bohr, V. A., & Souza-Pinto, N. C. de. (2010). Mitochondria and aging (Editorial). Mechanisms of Ageing and Development. Clare: Instituto de Química, Universidade de São Paulo. doi:10.1016/j.mad.2010.07.002
NLM
Prolla TA, Bohr VA, Souza-Pinto NC de. Mitochondria and aging (Editorial) [Internet]. Mechanisms of Ageing and Development. 2010 ; 131( 7-8): 449-450.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.mad.2010.07.002
Vancouver
Prolla TA, Bohr VA, Souza-Pinto NC de. Mitochondria and aging (Editorial) [Internet]. Mechanisms of Ageing and Development. 2010 ; 131( 7-8): 449-450.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.mad.2010.07.002
Artigo de periodico
Mitochondrial base excision repair assays (2010)
Maynard, Scott; Souza-Pinto, Nadja Cristhina de ; Scheibye-Knudsen, Morten; Bohr, Vilhelm A
Source: Methods. Unidade: IQ
Subjects: MITOCÔNDRIAS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAYNARD, Scott et al. Mitochondrial base excision repair assays. Methods, v. 51, n. 4, p. 416-425, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.ymeth.2010.02.020. Acesso em: 28 abr. 2024.
APA
Maynard, S., Souza-Pinto, N. C. de, Scheibye-Knudsen, M., & Bohr, V. A. (2010). Mitochondrial base excision repair assays. Methods, 51( 4), 416-425. doi:10.1016/j.ymeth.2010.02.020
NLM
Maynard S, Souza-Pinto NC de, Scheibye-Knudsen M, Bohr VA. Mitochondrial base excision repair assays [Internet]. Methods. 2010 ;51( 4): 416-425.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.ymeth.2010.02.020
Vancouver
Maynard S, Souza-Pinto NC de, Scheibye-Knudsen M, Bohr VA. Mitochondrial base excision repair assays [Internet]. Methods. 2010 ;51( 4): 416-425.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.ymeth.2010.02.020
Artigo de periodico
Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions (2010)
Liu, Dong; Croteau, Deborah L; Souza-Pinto, Nadja Cristhina de ; Pitta, Michael; Tian, Jingyan; Wu, Christopher L; Jiang, Haiyang; Mustafa, Khadija; Keijzers, Guido; Bohr, Vilhelm A; Mattson, Mark Paul
Source: Journal of Cerebral Blood Flow & Metabolism. Unidade: IQ
Subjects: DANO AO DNA, MITOCÔNDRIAS
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ABNT
LIU, Dong et al. Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions. Journal of Cerebral Blood Flow & Metabolism, v. 31, n. 2, p. 680-692, 2010Tradução . . Disponível em: https://doi.org/10.1038/jcbfm.2010.147. Acesso em: 28 abr. 2024.
APA
Liu, D., Croteau, D. L., Souza-Pinto, N. C. de, Pitta, M., Tian, J., Wu, C. L., et al. (2010). Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions. Journal of Cerebral Blood Flow & Metabolism, 31( 2), 680-692. doi:10.1038/jcbfm.2010.147
NLM
Liu D, Croteau DL, Souza-Pinto NC de, Pitta M, Tian J, Wu CL, Jiang H, Mustafa K, Keijzers G, Bohr VA, Mattson MP. Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions [Internet]. Journal of Cerebral Blood Flow & Metabolism. 2010 ; 31( 2): 680-692.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/jcbfm.2010.147
Vancouver
Liu D, Croteau DL, Souza-Pinto NC de, Pitta M, Tian J, Wu CL, Jiang H, Mustafa K, Keijzers G, Bohr VA, Mattson MP. Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions [Internet]. Journal of Cerebral Blood Flow & Metabolism. 2010 ; 31( 2): 680-692.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/jcbfm.2010.147
Artigo de periodico
DNA repair and the accumulation of oxidatively damaged DNA are affected by fruit intake in mice (2010)
Croteau, Deborah L; Souza-Pinto, Nadja Cristhina de ; Harboe, Charlotte; Keijzers, Guido; Zhang, Yongqing; Becker, Kevin; Sheng, Shan; Bohr, Vilhelm A
Source: Journal of Gerontology: Biological Science. Unidade: IQ
Subjects: DANO AO DNA, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CROTEAU, Deborah L et al. DNA repair and the accumulation of oxidatively damaged DNA are affected by fruit intake in mice. Journal of Gerontology: Biological Science, v. 65, n. 12, p. 1300-1311 : + Supplementary materials, 2010Tradução . . Disponível em: https://doi.org/10.1093/gerona/glq157. Acesso em: 28 abr. 2024.
APA
Croteau, D. L., Souza-Pinto, N. C. de, Harboe, C., Keijzers, G., Zhang, Y., Becker, K., et al. (2010). DNA repair and the accumulation of oxidatively damaged DNA are affected by fruit intake in mice. Journal of Gerontology: Biological Science, 65( 12), 1300-1311 : + Supplementary materials. doi:10.1093/gerona/glq157
NLM
Croteau DL, Souza-Pinto NC de, Harboe C, Keijzers G, Zhang Y, Becker K, Sheng S, Bohr VA. DNA repair and the accumulation of oxidatively damaged DNA are affected by fruit intake in mice [Internet]. Journal of Gerontology: Biological Science. 2010 ; 65( 12): 1300-1311 : + Supplementary materials.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1093/gerona/glq157
Vancouver
Croteau DL, Souza-Pinto NC de, Harboe C, Keijzers G, Zhang Y, Becker K, Sheng S, Bohr VA. DNA repair and the accumulation of oxidatively damaged DNA are affected by fruit intake in mice [Internet]. Journal of Gerontology: Biological Science. 2010 ; 65( 12): 1300-1311 : + Supplementary materials.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1093/gerona/glq157

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